![]() ![]() ![]() Alicia Borinsky s voice, her words that planted the seeds of this research Te interesará ver las tapas de Claridad con los escritores rusos are also the ones that led me to discover the core of the dissertation: our shared way back to Russia, inevitably, from within Buenos Aires. Solo uno ivĦ ACKNOWLEDGMENTS Many languages, voices, and chance acquaintances make up each page of this dissertation. And to the one step: De las pampas argentinas a las estepas rusas, tan solo un paso. Assistant Professor of Latin American Artĥ DEDICATION To my parents, Nelya and Ramil, who, between Asia and Europe, once made and are still making it all possible. ![]() Assistant Professor of Spanish Third Reader Ana María Reyes, Ph.D. Professor of Spanish and Latin American Studies Second Reader Rodrigo Lopes de Barros, Ph.D. 1 Boston University OpenBU Theses & Dissertations Boston University Theses & Dissertations 2017 Translation as cultural contraband: translating and writing Russian literature in Argentina Fauzetdinova, Adel Ramilevna Boston UniversityĢ BOSTON UNIVERSITY GRADUATE SCHOOL OF ARTS AND SCIENCES Dissertation TRANSLATION AS CULTURAL CONTRABAND: TRANSLATING AND WRITING RUSSIAN LITERATURE IN ARGENTINA OR HOW BAD TRANSLATIONS MADE GOOD LITERATURE by ADEL RAMILEVNA FAUZETDINOVA B.A., Framingham State University, 2011 M.A., Boston University, 2013 Submitted in partial fulfillment of the requirements for the degree of Doctor of Philosophy 2017ģ 2017 by ADEL RAMILEVNA FAUZETDINOVA All rights reservedĤ Approved by First Reader Alicia Borinsky, Ph.D. ![]()
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![]() CXCL12 protein levels were reduced in plaque and peritoneal macrophages in MF-IGF1 mice. Monocyte recruitment was reduced by 70% in MF-IGF1 mice and was associated with a 27% reduction in circulating levels of CXC Chemokine Ligand 12 (CXCL12). Macrophage IGF1 overexpression downregulated plaque burden by 30%, reduced plaque macrophages by 47%, and promoted features of a stable plaque phenotype. We also assessed cholesterol efflux and foam cell formation in vivo and in vitro. ![]() We accelerated atherosclerotic development by feeding animal a high fat diet for three months. We hypothesized that increasing IGF1 levels strictly in macrophages will prevent atherosclerosis.Īfter breeding a novel macrophage-specific IGF1 overexpressing transgenic mouse to an Apoe -/- background (MF-IGF1 mice), we assessed atherosclerotic plaque burden, stability, and monocyte recruitment. Results of our previous in vitro experiments suggest that macrophages play a predominant role in mediating IGF1 effects in atherosclerotic plaque, but exact mechanisms remain unclear. ![]() ![]() IGF1 administration in ApoE deficient (Apoe -/-) mice fed a high fat diet reduced atherosclerosis and reduced plaque macrophages. Insulin Like Growth Factor I (IGF1) has been shown to reduce cardiovascular events. 1Tulane University School of Medicine, New Orleans, LAĢUniversity of Missouri System, Columbia, MOĪtherosclerosis is the leading cause of Cardiovascular Disease, which is still the global leader of mortality. ![]() ![]() ![]()
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